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2021, Accessed 20 Nov

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Further exploration of how targeting NF-B signaling interrupts EBV latency in NPC cells might reveal new options for NPC treatment

7 Collectively, this study suggested that the tumor microenvironment can deliberately manipulate neutrophil reprogramming, transforming immature (T1) and mature (T2) neutrophils into the pro-tumorigenic T3 phenotype, capable of promoting tumor proliferation and metastasis

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